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. 1998 Nov 1;18(21):8551–8558. doi: 10.1523/JNEUROSCI.18-21-08551.1998

Fig. 5.

Fig. 5.

Potentiation of NO release induced by forskolin (FSK) and a cAMP analog. A, Potentiation of NO release induced by 50 μmforskolin (filled circles).Inset shows superposed traces recorded before application of forskolin (a), in the presence of forskolin (b), and 30 min after the cessation of forskolin application (c). Forskolin-induced potentiation in the presence of 10 μmH89 is also shown (open circles). In this experiment, slices were incubated with 10 μm H89 for at least 2 hr before and throughout the recording. B, Potentiation of NO release induced by forskolin and TS (filled circles). Inset shows tracesrecorded before application of forskolin (a), before initiation of TS (b), and 30 min after the cessation of forskolin application and TS (c). Changes in NO release elicited by application of 1,9-dideoxyforskolin (50 μm, DideoxyFSK) are also shown (open circles). C, Potentiation of NO release induced by 1 mm Br-cAMP (horizontal bar). D, Effect of 1 mm Br-cGMP (horizontal bar) on NO release.