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. 2017 Mar 7;7:43199. doi: 10.1038/srep43199

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Copyright © 2017, The Author(s)

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(A) The A. parvulum and P. copri -induced signal were inhibited using the specific inhibitor for MEK/ERK (U0126) on HT-29-PPARγ cells. Different letters indicate statistically different results (p < 0.05). (B) The implication of MEK/ERK in PPARγ activation by A. parvulum and P. copri was confirmed on the protein level showing phosphorylation of ERK1/2. Total ERK1/2 was used as control for phosphorylated PPARγ. GAPDH was used as loading control.