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. 2015 May 26;113(1):46–56. doi: 10.1038/bjc.2015.186

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Copyright © 2015 Cancer Research UK

From twelve months after its original publication, this work is licensed under the Creative Commons Attribution-NonCommercial-Share Alike 4.0 Unported License. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-sa/4.0/

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DNA damage and apoptosis in SK-LMS-1 and HT1080 xenografts. Graphs demonstrating the number of CD31+/γH2AX+/cleaved caspase-3+ cells (A) and CD31−/γH2AX+/cleaved caspase-3+ cells (B) in SK-LMS-1 tumours from each treatment group. (C) Immunofluorescence photos of tumours from each treatment group after staining for CD31 (green), γH2AX (white), cleaved caspase-3 (red), and nuclei (DAPI, 4',6-diamidino-2-phenylindole). Graphs demonstrating the number of CD31+/γH2AX+/cleaved caspase-3+ cells (D) and CD31−/γH2AX+/cleaved caspase-3+ cells (E) in HT1080 tumours from each treatment group. (E) Immunofluorescence photos of tumours from each treatment group after staining for CD31 (green), γH2AX (white), cleaved caspase-3 (red), and nuclei (DAPI). Bars represent standard deviation. Scale bar, 20 μm. **P<0.05 compared with all other groups.