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. 2014 Jun 18;35(8):1680–1690. doi: 10.1093/carcin/bgu134

Fig. 1.

Fig. 1.

The complex cross talk between p53 and NF-κB. Both transcription factors respond to a set of activating signals that accumulate and determine the interaction that varies from profound corepression to mutual activation, highly dependent on the cellular context.