Skip to main content
. Author manuscript; available in PMC: 2010 Apr 21.
Published in final edited form as: Int J Cardiol. 2008 Jul 25;130(2):147–158. doi: 10.1016/j.ijcard.2008.04.059

Figure 1.

Figure 1

Ischemia of sufficient duration to induce myocardial infarction stimulates an inflammatory reaction that is characterized by infiltration of neutrophils during the first day and macrophages beginning at day 3. The influx of macrophage coordinates the wound healing response, which involves activation of both endothelial cells and cardiac fibroblasts. The coordination between inflammation and scar formation determines the fate post-MI.