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. Author manuscript; available in PMC: 2010 Mar 13.
Published in final edited form as: Neurosci Lett. 2009 Jan 23;452(2):204–208. doi: 10.1016/j.neulet.2009.01.049

Fig. 1.

Fig. 1

A, B) T2 weighted images, 0.5 mm thick, coronal, and centered at −1.0 mm (A) and −1.5 mm (B) relative to Bregma from an aged C57BL/6 mouse (~24 months) given a CCI injury 2 months previously. The primary injury site appears as a hyperintensity in the sensorimotor cortex. T2 hypointense areas are present in the ipsilateral thalamus (arrowheads) and ipsilateral internal capsule (arrow) relative to the contralateral structures. C) Abnormal iron deposits (arrowheads), revealed by iron histochemistry, co-localize to the T2 hypointensity in the thalamus (the same animal as for A, B). Bar = 2 mm (A–C).