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A nonredundant role of α4β1 and αLβ2 in lymphoid progenitor homing to the thymus. lin− BM cells from pTα-hCD25 mice were pretreated with anti-α4, anti-β2, both, or control mAbs and adoptively transferred into Rag2−/−γc−/− mice treated or not with anti-VCAM-1 or anti-ICAM-1 mAbs. Twenty hours later, thymi (A) and BM (B) were harvested, and cell suspensions were stained for Ly5.1 and hCD25 (n = 4 mice).
