Skip to main content
. 2026 Sep 29;208(10):e00360-26. doi: 10.1128/jb.00360-26

Fig 1.

Diagram of A. baumannii showing the key mechanisms of antibiotic resistance, stress responses driving persistence in hopital settings (including dessication and oxidative stress tolerance), genomic plasticity, and virulence.

The toolkit of a successful opportunistic pathogen. A. baumannii relies on a limited yet highly integrated set of traits that collectively enable persistence in clinical environments and opportunistic infection. These include antibiotic resistance (A), stress responses involved in the success of A. baumannii in hospital settings (B), genomic plasticity (C), and host interaction and virulence strategies (D). aa, amino acids; ATB, antibiotics; IS, insertion sequences; LOS, lipooligosaccharide; PAA, phenylacetic acid; T1SS, type 1 secretion system; T2SS, type 2 secretion system; TCS, two-component systems; Tn, transposons.